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At first glance, it reads like a familiar digital health curiosity: rapid growth, an unconventional operating model, and a cluster of lawsuits that seem to follow in the wake of success

The mechanism is direct: SS-31 improves mitochondrial ATP production efficiency, and cellular fatigue is substantially a function of insufficient ATP availability

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Our findings identify NKX21 tumours as an extra subset of cancers that could benefit from the therapeutic targeting of serine/glycine synthesis hyperactivation with drugs such as sertraline